Genome-Wide Association Study for Type 2 Diabetes in Indians Identifies a New Susceptibility Locus at 2q21

نویسندگان

  • Rubina Tabassum
  • Ganesh Chauhan
  • Om Prakash Dwivedi
  • Anubha Mahajan
  • Alok Jaiswal
  • Ismeet Kaur
  • Khushdeep Bandesh
  • Tejbir Singh
  • Benan John Mathai
  • Yogesh Pandey
  • Manickam Chidambaram
  • Amitabh Sharma
  • Sreenivas Chavali
  • Shantanu Sengupta
  • Lakshmi Ramakrishnan
  • Pradeep Venkatesh
  • Sanjay K. Aggarwal
  • Saurabh Ghosh
  • Dorairaj Prabhakaran
  • Reddy K. Srinath
  • Madhukar Saxena
  • Monisha Banerjee
  • Sandeep Mathur
  • Anil Bhansali
  • Viral N. Shah
  • Sri Venkata Madhu
  • Raman K. Marwaha
  • Analabha Basu
  • Vinod Scaria
  • Mark I. McCarthy
  • Radha Venkatesan
  • Viswanathan Mohan
  • Nikhil Tandon
  • Dwaipayan Bharadwaj
چکیده

Indians undergoing socioeconomic and lifestyle transitions will be maximally affected by epidemic of type 2 diabetes (T2D). We conducted a two-stage genome-wide association study of T2D in 12,535 Indians, a less explored but high-risk group. We identified a new type 2 diabetes-associated locus at 2q21, with the lead signal being rs6723108 (odds ratio 1.31; P = 3.32 × 10⁻⁹). Imputation analysis refined the signal to rs998451 (odds ratio 1.56; P = 6.3 × 10⁻¹²) within TMEM163 that encodes a probable vesicular transporter in nerve terminals. TMEM163 variants also showed association with decreased fasting plasma insulin and homeostatic model assessment of insulin resistance, indicating a plausible effect through impaired insulin secretion. The 2q21 region also harbors RAB3GAP1 and ACMSD; those are involved in neurologic disorders. Forty-nine of 56 previously reported signals showed consistency in direction with similar effect sizes in Indians and previous studies, and 25 of them were also associated (P < 0.05). Known loci and the newly identified 2q21 locus altogether explained 7.65% variance in the risk of T2D in Indians. Our study suggests that common susceptibility variants for T2D are largely the same across populations, but also reveals a population-specific locus and provides further insights into genetic architecture and etiology of T2D.

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عنوان ژورنال:

دوره 62  شماره 

صفحات  -

تاریخ انتشار 2013